Tag Archives: Respiratory-to-glycolytic Shift

Glycolysis Study of 236 Patients With Dysoxygenosis

                                           Majid Ali, M.D. In 1998, I coined the term dysoxygenosis for a state of dysfunctional oxygen metabolism. The glycolysis data presented below was obtained with one of my several studies on the subject of ATP generation. For professional readers interested in this subject, I suggest the 10th, 11th, and 12th volumes of my textbook entitled “The Principles and Practice

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Crucial Enzymes in the Oxygen Order of Human Health – Oxyenzymes

  Oxyenzymes Majid Ali, M.D.  In this tutorial , I present fundamrentals of enzymes of central importance in the free radical pathology and immunology. Myeloperoxidase Deficiency It is of some interest to note that the essential role of eukocyte myeloperoxidase in preserving the function integrity of the phagocytes was first demonstrated in disseminated candidiasis in 1969 by Lehrer and Cline.

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Fermentation – Good, Bad, and Ugly

                                                    Majid Ali, M.D. Our early primordial ancestors were fermenters. Throughout human evolution, some of those fermenting cells thrived in oxygen-poor nitches in the human body, serving many purposes, including food digestion. These were “good fermenters.” Our later human ancestors learned—experientially or intuitively, it seems—learned ferment foods to enhance their value. We can call it “good fermentation.” Every chronic

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Respiratory-to-Fermentative Shift in ATP Production

                                                                          Majid Ali, M.D.                                                                            August 2014 Syndromes of persistent and debilitating fatigue— fibromyalgia, chronic fatigue syndrome, persistent fatigue following chemotherapy for malignant disorders, and others — may be properly designated “chronic energy deficit states.” There is an enormous body of literature concerning clinical patterns, symptom-complexes, and putative etiologic agents. A recent (February/March) issue of Townsend Letters was devoted to

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