The Eclampsia-Autism Spectrum Link


Majid Ali, M.D.

Eclampsia, also called toxemia of pregnancy, is fundamentally an oxygen problem caused by toxicities of food, environment, and thought. Autism is rooted in toxicities of the womb which impaired oxygen functions in the brain development. That, simply stated is the essential connection between the two.

On Feb. 20, 2015, MedScape Pathology & Lab Medicine reported on a study that provides a clear proof of the above.

The conclusion of the study was exactly what the Oxygen Model predicts. Consider the following text from the Medscape article: “Results from the Northern California-based Childhood Autism Risks from Genetics and the Environment (CHARGE) study show that exposure to preeclampsia in utero was associated with a greater-than-twofold increased risk for ASD and a greater-than-fivefold increased risk for developmental delay, compared with no exposure.”

How Did the Oxygen Model of Autism Predict the Preeclampsia-Autism Connection?

The commonly used term for eclapsia is toxemia of pregnancy. The pathology of eclapsia is well established and the core commonality in all its clinical features is fetal and maternal tissue injury caused by lack of oxygen. It is that simple. Preeclapsia, of course, “pre-toxemia,” a stage just before toxemia of pregnancy.

So the simple and straightforward answer to the question in the above subtitle above is self-evident.

Simply stated, the Oxygen Model of Autism states that the fundamental cause of the autism spectrum disorder is dysfunctional oxygen metabolism, predominantly developing as a consequence toxic states in the womb. Preeclapsia is precisely that: lack of adequate and functional oxygen for the unborn baby due to toxic womb conditions.

The Oxygen Models of Preeclampsia and Autism

The Oxygen Model of Autism and The Oxygen Model of Preeclampsia are both extensions of my Oxygen Model of Health and Disease. They are unifying models that explain all aspects of the two disorders—causes, clinical course, consequences, and control—on the basis of disturbed oxygen function. The most important among these compromised and/or blocked functions are: (1) oxygen signaling; (2) oxygen’s ATP energy generation; (3) oxygen’s detergent functions; (4) oxygen’s cellular detox functions; (5) oxygen-regulated cell membrane and matrix functions; (6) oxygen’s cellular repair roles.

The Oxygen Model of Autism and The Oxygen Model of Preeclampsia provide simple models that allows physicians to reduce complexities of diverse clinical syndromes into a workable simplicity.

These models predict that ongoing research will reveal that components of acidosis (excess acidity), oxidosis (increased oxidative stress), and CUD (clotting-unclotting dysequilibrium) will be found to play important roles in the pathology and clinical features of preeclamsia and autism.

The crucial importance of the Unifying Oxygen Model of Preeclampsia and Autism are that they:

* Explain the scientific basis of primary mechanism of cellular energetic dysfunction in the two disorders;

* Shed light how health can be restored by addressing all relevant oxygen-related issues;

* Elucidate how toxicities of foods, environments, and thoughts cause tissue injury and disease;

* Reveal the mechanisms by which various detox therapies work (Oxygen is the primal detergent which removes cellular grease and allows cells to breathe freely).

* Allow the formulation of rational and effective designs for preventing, arresting, and reversing these disorder; and

* Provide explanations of mechanisms by which time-honored natural remedies work.

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