The Diabetes-Dementia Connection – The Oxygen Link

Majid Ali, M.D.

Is there a connection between diabetes and dementia? If so, what is that connection? These are the two questions I received from a reader. I thank that reader. Such letters allow me to enhance the value of video and textual parts of my Science, Health, and Healing Encyclopedia.

Yes, there is a connection between diabetes and dementia. What connects the two diseases is oxygen, dysfunctional oxygen metabolism (dysox for short, pronounced as ‘dis’ and ‘ox’). What makes these questions especially disturbing is that both diseases are spreading as pandemics.

Dysox Links Diabetes and Dementia

Simply stated, both diabetes and dementia at their roots are oxygen problems and caused by dysox. Indeed, the link between the two diseases is predicted by my oxygen models of the two diseases. Below, I present these oxygen models for readers with deep interest in the subject. First, I cite a recent study which defines the extent of this relationship in a numerical sense.

In December 2014, Professor Elizabeth Selvin at the Johns Hopkins Bloomberg School of Public Health and her colleagues published a report in the Annals of Internal Medicine. After adjusting for several related health and behavioral factors, they found people with diabetes suffered a 30 percent larger decline in mental acuity than those without the disease.

The authors suggested that the association of diabetes with mentation and memory problems were the result of damage to small blood vessels in the brain. I agree with that conclusion. However, this is a very superficial view of the problem, the real nature of which at the deeper bio-energetic level becomes evident from a consideration of the oxygen models of the two diseases reproduced below from previous writings on the subjects.

My Oxygen Models of Diabetes and Dementia

The Oxygen Model of Diabetes and The Oxygen Model of Dementia are both extensions of my Oxygen Model of Health and Disease. They are unifying model that explains all aspects of the two diseases—causes, clinical course, consequences, and control—on the basis of disturbed oxygen function. The most important among these compromised and/or blocked functions are: (1) oxygen signaling; (2) oxygen’s ATP energy generation; (3) oxygen’s detergent functions; (4) oxygen’s cellular detox functions; (5) oxygen-regulated cell membrane and matrix functions; (6) oxygen’s cellular repair roles.

The Oxygen Models of Diabetes and The Oxygen Model of Dementia provide simple models that allows physicians to reduce complexities of diverse clinical syndromes into a workable simplicity.

This model predicts that ongoing research will reveal that components of acidosis (excess acidity), oxidosis (increased oxidative stress), and CUD (clotting-unclotting dysequilibrium) will be found to play important roles.

The crucial importance of the Unifying Oxygen Model of Diabetes and Dementia are that they:

Explain the scientific basis of primary aging processes in the body;

Shed light how health can be preserved by addressing all oxygen-related issues;

Elucidate how toxicities of foods, environments, and thoughts cause tissue injury and disease;

Reveal the mechanisms by which various detox therapies work (Oxygen is the primal detergent which removes cellular grease and allows cells to breathe freely).

Allow the formulation of rational and effective designs for reversing chronic diseases; and

Provide explanations of mechanisms by which time-honored natural remedies work.

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